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Support for an alternative to the classical proton gate characteristic of this HCO class is presented.
2
Natural organic matter and HCO 3 - were the main responsible for reducing CAP degradation in STP effluent.
3
A correlation was also demonstrated between the absence of the indel and the expression of the Hco-acr-8b transcript.
4
The purpose of this study was to examine the role of vagal-cholinergic regulation on human proximal duodenal mucosal HCO-3 secretion.
5
Conclusion: HCoVs could play significant role in causing upper respiratory tract infections among adults and older children in rural areas of Ghana.
6
The Hco-acr-8b transcript contains exons 1 and 2 and terminates with 347bp from within the intron 2.
7
The response to sham feeding was approximately 50% of the peak response to acid-stimulated HCO-3 output.
8
HCOs function as the terminal enzymes in the respiratory chain of mitochondria and aerobic prokaryotes, coupling molecular oxygen reduction to transmembrane proton pumping.
9
These findings indicate that basal human proximal duodenal mucosal HCO-3 secretion is maintained largely by resting cholinergic innervation and is stimulated by cephalic-vagal stimulation.
10
Among all HCOs, those made of intrinsic irregular neurons presented a wider burst frequency range while keeping a reliable regular oscillatory (bursting) behavior.
11
We also discuss how HCO-3 interacts with CFTR as both a permeating anion and a potential regulator of Cl- permeation through the CFTR ion channel.
12
Two-neuron CPG, half center oscillator (HCO), was obtained for each intrinsic behavior of the neurons by coupling them with mutual symmetric synaptic inhibition.